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These results suggest that overall the p16 ink4a gene is more responsible in inducing cellular senescence and tumor suppression in human cells interestingly the ink4aarf locus is known to be repressed by the polycomb proteins b lymphoma momlv insertion region 1 bmi1 and related family members in both human and mouse cellscellular senescence and tumor suppression it is now clear that cellular senescence is a crucial anticancer mechanism that prevents the growth of cells at risk for neoplastic transformation the stimuli that elicit a senescence response all have the potential to initiate or promote carcinogenesisnormal aging aging can be defined as the decline and deterioration of functional properties at the cellular tissue and organ level this loss of functional properties yields a loss of homeostasis and decreased adaptability to internal and external stress yielding an increased vulnerability to disease and mortality aging is a breakdown in maintenance of specific molecular structures and cellular senescence and tumor suppression editors peter d adams cruk beatson labs university of glasgow garscube estate switchback road glasgow g20 8pu uk padamsbeatsonglaacuk john m sedivy department of molecular biology cell biology and biochemistry brown universitycellular senescence and tumor suppression by peter d adams read online or download in secure pdf format

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